One: Introduction to the sequestration hypothesis. Two: The sampling theory of fibrotic arteriosclerosis. Three: Intrusion of atheroma into the most fibrotically thickened intimal sites. Four: Conditions for the intrusion of atheroma in coronary artery. Five: The size of the SMC realm assessed with the help of sampling theory. Six: Biased censoring of low SMC sites by atheroma in coronary artery. Seven: Biased sampling of low SMC sites by atheroma in thoracic aorta. Eight: SMC numbers at varying depths in intima of thoracic aorta. Nine: Histologic appearances of SMC clusters and realms. Ten: Direct imaging of the hypothetical quantity, sequestered lipid. Eleven: Local sequestration of lipid from place to place within an artery. Twelve: Fibroplasia in microscopic renal arteries. Thirteen: Parameters of fibroplasia in renal microvasculature. Fourteen: The course of arterial intimal fibroplasia in aging arteries. Fifteen: The course of fibroplasia per SMC over time in aging arteries. Sixteen: Fibroplasia per SMC in the media of coronary arteries. Seventeen: Influence of arteriolar hyalinization on renovascular fibroplasia. Eighteen: The Hy effect on Ra in widely variable circumstances. Nineteen: Hyalinized renal arterioles and the maleness coronary risk factor. Twenty: Two pathways to atheroma variably linked to renovasculopathies. Twenty One: Age of onset of the sex difference in coronary fibroplasias. Twenty Two: Adrenocortical nodularity in relation to coronary fibroplasia. Twenty Three: Atheroma and intimal fibroplasia in periodontal disease. Twenty Four: Atheroma and intimal fibroplasia in relation to obesity. Twenty Five: Paucity of literature relevant to SMC numbers and the aging risk factor.